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Citing this page: Bosc, N. and Lefranc, M.-P., Exp. Clin. Immunogenet., 18, 51-58 (2001) PMID: 11150853 pdf

"+" or "-" indicates if the gene sequences have been found (+) or not been found (-) rearranged (R), transcribed (T) and/or translated into protein (Pr). Arbitrarily that information is shown on the first line of each gene when the data have been confirmed by several studies.

Functionality is shown between parentheses, (F) and (P), when the accession number refers to rearranged genomic DNA or cDNA and the corresponding germline gene has not yet been isolated.
Functionality is shown between brackets, [F] and [P], when the accession number refers to genomic DNA, but not known as being germline or rearranged.

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IMGT gene name IMGT allele name Fct Chromosomal localization R T Pr Positions in the locus IMGT/LIGM-DB reference sequences IMGT/LIGM-DB sequences from the literature
Strain Clone names Accession numbers Positions in the sequence Secondary accession numbers Strain Clone names Accession numbers Positions in the sequence
TRDJ1 TRDJ1*01 F 14 (19.7 cM) + + + BALB/c (3) Jdelta 1 AF019412 [2]
TRDJ2 TRDJ2*01 F 14 (19.7 cM) + + + BALB/c Jdelta 2 MAP X64903 [1] B10.D2-H2dm1 (1) M64239 [4]
TRDJ2*02 F (2) 14 (19.7 cM) Std:ddY Jdelta 2 X17179 [3]

MAP: Mapped sequences.

IMGT notes:
  1. (1) B10.D2-H2dm1 is a congenic strain, B10 is the abbreviated symbol of C57BL/10 and D2 the abbreviated symbol of DBA/2J.
  2. (2) Nucleotide T, at position 24 (according to the IMGT allele mutation numbering) of TRDJ2 in X64903, is deleted in X17179. This DELETION leads to a frameshift downstream of codon 8. However it is not excluded that V-D-J rearrangements allow to reestablish the normal ORF. For these reasons the TRDJ2*02 is considered as functional.
  3. (3) It is not excluded that the source of the sequence is from B10.D2-H.2dm1 instead of BALB/c, as the cosmid clone is coming from the Kai Wang's library [4].
IMGT references:
  1. [1] Chien Y.H. et al., Nature, 330, 722-727 (1987). PMID: 2961997
  2. [2] Rowen L. et al., Unpublished (1997).
  3. [3] Toda M. et al., J. Mol. Biol., 202, 219-231 (1988). PMID: 3172216
  4. [4] Wilson R.K. et al., Genomics, 13, 1198-1208 (1992). PMID: 1505953
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